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Decompression Illness
During a dive, nitrogen from the compressed air that the diver
breathes is dissolved into the blood and body tissues in accordance to
Henry’s law. When the diver returns to the surface, nitrogen may come
out of solution in the form of bubbles, due to the high gradient between
the partial pressures of the nitrogen in the tissues and the
environment.
The manifestations of DCS depend on the organs or tissues affected by
bubble formation. The commonest presentations of DCS are generalized
symptoms of weakness, tiredness, apathy and malaise. Musculoskeletal
symptoms presents with pain, and limitation of movement of the affected
joint. Neurological DCS presentations are varied with involvement of the
cerebral, cerebellar, inner ear, vision and peripheral nerves. DCS may
also present with cardiorespiratory and cutaneous involvement.
More recently, an umbrella term ‘acute decompression illness’ (DCI)
is used for both DCS and Cerebral Arterial Gas Embolism (CAGE), as their
presentation and treatment is similar.
Recompression treatment is the definitive treatment of both DCS and
CAGE. Delay increases both morbidity and mortality. The increase in
pressure during treatment reduces the bubble size (Boyle’s law), while
oxygen hastens bubble resolution by enhancing the diffusion gradient
between the bubble and its surroundings. |